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Monomolecular dual- or triagonists that engage GLP-1R, the glucagon receptor (GCGR), and/or the glucose-dependent insulinotropic polypeptide receptor (GIPR) extract additive metabolic and weight loss benefits over targeting the GLP-1R alone for the broad treatment of cardiometabolic disorders
282 These cytokines sensitize nociceptive neurons, leading to neuropathic pain, and promote neuronal damage through demyelination and axonal degeneration
Better fat loss than single agents
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